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Male pattern hair damage (androgenetic alopecia) is the commonest reason behind hair damage in men. Alopecia areata is a patchy hair loss that is usually of abrupt onset and can occur on any hairy section of the body. It is a non-scarring hair type loss with no obvious skin disease or lesion or other root systemic disturbance. Most cases entail the scalp or facial beard and rarely it may almost all of the body where it is known as alopecia universalis. Complete loss of scalp hair is called peladera totalis, or more especially as alopecia capitis totalis.
Alopecia areata has a tendency to occur most often in adults 30 to 60 years of age. Yet , it can also affect older people and, rarely, young children. Alopecia areata is not really contagious It should become distinguished from hair dropping that may occur following a discontinuation of hormonal progesterone and estrogen therapies for birth control or the hair shedding linked with the end of pregnancy There are a number of treatable circumstances that could be confused with alopecia areata.
Photochemotherapy — In photochemotherapy, the person is given a light-sensitive medicine (either placed on the skin or taken by mouth) and then exposed to an ultraviolet light supply. Studies have shown inconsistant results. However, a trial of photochemotherapy may be reasonable in people with extensive alopecia areata in the event topical immunotherapy is certainly not available. Treatment is generally ongoing for four to 6 months.
These are good examples of possible biochemical means by which emotional phenomena could influence the genesis of alopecia areata lesions through the action of neuromediators. Furthermore, in these types of types of disease, in that the illness itself generates evident psychological alterations through detriment towards the self-image, this is necessary to consider not only the possibility of emotional phenomena interfering in the disease, but that the disease itself can certainly produce important psychological modifications.
Male androgenetic alopecia is usually genetically susceptible, and no additional exploration is necessary. Female androgenetic alopecia often appears in women with a good family history of hairloss or a personal good hirsutism, acne, or abnormal menses (signs of vom männlichen geschlechtshormon excess). Genetically predisposed ladies may present with androgenetic alopecia in adolescence (puberty), perimenopause, or postmenopause. Youthful women have a larger incidence of acquired well known biotebal dawkowanie adrenal hyperplasia and polycystic ovaries. Generally, postmenopausal women possess lower levels of human hormones, especially estrogen. However, androgenic hormone or testosterone levels in postmenopausal females are relatively high when compared with levels in adolescents. Androgen excess screening for women with curly hair loss includes measurements of total testosterone and dehydroepiandrosterone sulfate.

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